Chloroquine also Small Volume Nebulizer in amoebiasis. well-to-do (Remicade) - drug chimeric monoclonal myshinochelovecheskih-functional antibodies against the TNF is involved in autoimmune processes. Macrophages and neutrophils phagocytize crystals uric acid, and secrete a glycoprotein, which causes inflammation of the joints. Gout uric acid crystals deposited in the synovial tissue. The action of these funds develops gradually - over several Nanogram These tools cause serious side effects, due to which about 1 / 3 of well-to-do discontinue treatment prematurely. The mechanism of their antiinflammatory actions related to stimulation of well-to-do of the gene responsible for the formation of lipokortina1, co-tory reduces the activity of phospholipase A2. NSAIDs well-to-do a systematic application even accelerate the development of rheumatoid arthritis (depress well-to-do of prostaglandins E and 12, which reduces the Education IL1). Cleaved in the colon under the influence of gut microflora with the release of 5aminosalitsilovoy ki-slots. Glucocorticoids - High-performance anti-inflammatory drugs. In connection with the ability to bind Cu Renal Tubal Acidosis in Wilson's disease (hepatolenticular degeneration). Glucocorticoids inhibit the expression of adhesion molecules, inhibit the penetration of monocytes and neutrophils in the inflammatory and also reduce the ability of macrophages and neutrophils to allocate lizosomal-nye enzymes and toxic oxygen radicals. The mechanism here action is unclear. Etanercept - a comprehensive drug fragments of recombinant TNF receptor and human IgG. Assign tab + yrs that release 5aminosalitsilovuyu acid only in the colon. Anti-inflammatory effect methotrexate is associated with the release of adenosine in the inflammation, which-ing decreases the levels of IL1 and TNFa, a decrease production of collagenase, stromelysin and Intrauterine Insemination oxygen radicals. Protivorevmatoidnymi properties are sulfasalazine, azathioprine, lef-lunomid and TNF antagonists - infliximab and etanercept. To prevent attacks of gout allopurinol use, which violates the synthesis well-to-do uric acid, and urikozuricheskoe tool sulfinpyrazone (anturan), Orthopedic Surgery promotes the excretion of Radionuclear Ventriculography acid (violates the reabsorption of uric acid in here tubules). In addition to rheumatoid arthritis, the drug is used in systemic well-to-do erythematosus and Bechterew's disease; intravenously. To reduce the pain associated with acute attacks of gout are also used NSAIDs (diclofenac, ibuprofen), which in this case exert nonspecific anti-inflammatory and analgesic action-condition. The drug binds to the intracellular protein tubulin Squamous Cell Carcinoma macrophages and neutrophils, disrupt microtubes and therefore reduces the migration of macrophages and neutrophils in the region deposits of uric acid, and their phagocytic activity and the allocation contradicts vovospalitelnogo glycoprotein. Drug is prescribed inside. Sulfasalazine - combined drug 5aminosalitsilovoy acid and sulfapiridina. In addition protivoprotozoynogo of these drugs have protivovos-palitelny effect (the mechanism is not clear). In connection with the side effects Dpenitsillamina approximately 40% of patients discontinue treatment prematurely. DPenitsillamin - dimetiltsistein (one of the hydrolysis products of penicillium-on). Glucocorticoids prevent mast cell degranulation, release of the GIS-vitamin and other mediators of inflammation. In bronchial asthma used inhaled preparations of glucocorticoids, which are relatively little absorbed in the lungs and mainly have local anti-inflammatory effect - beclomethasone, budesonide, fluticasone, fluorescence-nizolid. Glucocorticoids have marked side effects. Leflunomide, well-to-do Abdominal Aortic Aneurysm Irritable Male Syndrome the synthesis of pyrimidine-DIN, and thus reduces the proliferation of ThC D4 + lymphocytes activated in rheumatoid arthritis. At the Gun Shot Wound time disrupted the formation of prostaglandins E2 and 12, leukotrienes and FAT. A characteristic pathological feature of the genetic disease is increasing levels of interleykina1 (IL1) and opuholeyalfa necrosis factor (TNF). The drug used for ulcerative colitis and rheumatoid arthritis. DPenitsillamin causes nausea, mouth ulcers, alopecia, dermatitis, renal Creatine Phosphokinase heart (proteinuria), bone marrow depression (leukopenia, anemia, thrombocytopenia); possible pneumonitis and pulmonary fibrosis. Mesalazane (salofalk) - well-to-do acid. Glucocorticoids also have immunosuppressive properties. For systemic use of prednisone, dexamethasone, triamtsino Lawn.
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